Anthrax, Bacillus Cereus, and Listeria: Characteristics, Pathogenesis, and Prevention

Bacillus anthracis:

Bacillus anthracis can enter through the skin, gastrointestinal tract, and the airway (less frequent but more lethal).

  • Gram-positive bacilli
  • Facultative anaerobe
  • Forms spores that can survive for years
  • Undemanding growth
  • Presents a polypeptide capsule (poly-D-glutamic acid)
  • Coded by 3 genes: capB, capC (Plasmid 1)
  • Plasmid 2 (B. anthracis virulence)

Encodes 3 Exotoxins:

  1. Protective antigen (binds to the host cell)
  2. Edema factor
  3. Lethal factor

Edema Toxin:

Edema factor + protective antigen = responsible for severe edema, a protective feature of infection.

Lethal Toxin:

Protective antigen + lethal factor.

Avirulent Strains:

Removing one or both plasmids can result in avirulent strains (harmless). Plasmid 1 encodes the capsule, and plasmid 2 encodes exotoxins.

Pathogenesis and Immunity:

Capsule inhibits phagocytosis. Edema toxin is an adenylate cyclase. Lethal toxin is a zinc metalloprotease, stimulating macrophages to release TNF-alpha and interleukin.

Routes of Infection in Humans:

Inoculation, ingestion, inhalation.

3 Cycles of B. Anthracis in Nature:

Multiplication of spores in the soil, food infection, human infection.

Types of Anthrax:

Cutaneous anthrax: Inoculation, 1-12 days, with itching and clear exudate.

Gastrointestinal anthrax: Inoculation, 1-7 days, with nonspecific symptoms (fever, nausea, abdominal pain).

Inhalation anthrax: Latency period of 2 months, asymptomatic (more lethal).

First Phase:

Nonspecific symptoms: fever, shortness of breath, cough, headache, vomiting, chills, abdominal pain, and chest pain.

Second Phase:

Worsening of symptoms, edema, significant mediastinal lymphadenopathy accompanied by symptoms of meningitis, shock, and death.

Prevention:

Vaccination of animals and people at risk (cattle workers). Infections must be incinerated to prevent sporulation.



Bacillus cereus:

  • Opportunistic pathogen
  • Low virulence
  • Gram-positive spore-forming
  • Facultative anaerobe
  • Undemanding growth

Risk Groups:

  • Consumption of contaminated food (rice, meat, vegetables, sauces)
  • Penetrating injuries in the eye
  • Intravenous injections
  • Hemolysin: RBC destruction
  • Lethal factor: Death occurs in animals.
  • Vascular permeability factor: Causes changes in blood vessels.
  • Necrotic toxin: Necrosis of the bowel.

Food Poisoning:

  • Emetic form: Consumption of rice contaminated with heat-resistant spores, incubation time 1-6 hours.
  • Diarrheal form: Consumption of contaminated meat, vegetables, or sauces.

LISTERIA

(Easy contamination of refrigerated food).

  • Disease in humans: Listeria monocytogenes
  • Small, gram-positive bacilli
  • Facultative anaerobe
  • Isolated, in pairs, or short chains
  • Mobile at room temperature
  • Stationary at 37°C

Risk Groups:

Neonates, elderly, pregnant women, immunocompromised individuals.

  • Introduced into the cell via internalins, interacting with surface glycoprotein receptors on the host cell.
  • Habitat: Ground, water, vegetation, intestinal contents of mammals, birds, and fish. 1-5% in healthy human carriers.