Depressants, Benzodiazepines, and Psychiatric Drugs: A Comprehensive Guide

Depressants

• Barbiturates

– Within the family of depressants (derived from barbituric acid)

– Used as sedative-hypnotic and antiepileptic medication

• Short-acting barbiturates (< 4 hours) – secobarbital, pentobarbital (Seconal, Nembutal)

• Intermediate (4 – 6 hours) – amobarbital (Amytal)

• Long (> 6 hours) – Mephobarbital (Mebaral), phenobarbital

• But à injection faster acting than oral administration

– Higher dose lasts longer than lower dose

• Ultra-short acting – mostly used for surgeries or emergency situations

– Little appeal as a recreational drug à not commonly abused

• In general, same effects as depressants

• Low doses (oral administration) – relaxation (and sense of euphoria)

– Disinhibition of the cerebral cortex

• Inhibitory influences from the cortex reduced

• Symptoms similar to inebriation or intoxication from low doses of alcohol

• Truth serum? Say anything serum?

• ↑ in dose levels, lower regions of the brain concerned with general arousal become affected

• 100 mg (therapeutic dose) – drowsy, sedated à Can affect performance of driving a car or operating heavy machinery

• Higher doses à hypnotic (sleep-inducing) effect

• Historically – used to treat insomnia

– Sleep induced not normal

– Suppressed REM (rapid eye movement) sleep

• REM sleep – dreaming and relaxation of the body

• If barbiturates taken then stopped, the body tries to compensate for lost REM sleep

– REM sleep rebound – longer periods of REM sleep

• Vivid upsetting nightmares à barbiturate hangover (next day)

• Groggy and “out of sorts”

• Possibility of lethal overdose

– Taking a high dose level (depends on tolerance)

– Taking with alcohol

à coma and death (excessive dose produces inhibition of the respiratory control centers in the brain)

• Alcohol + barbiturates è synergistic

• Suicidal potential à ↓ as prescription sedative

• Long-acting barbiturates

• Use as sleep medication initiates a cycle of behavior that can lead to dependence

• Even after brief use, can temporarily ↑ anxiety during the day à greater degree of insomnia than before

• Barbiturate-induced sleep à groggy the next morning à stimulant to feel alert à still feel stimulant at night à continue taking barbiturate to achieve sleep

• Tolerance

• Withdrawal symptoms à physical dependence

• Abrupt withdrawal can lead to death (closely resembles alcohol withdrawal)

• Because of problems associated with its treatment of insomnia, no longer used for this purpose

• Epileptic seizures; phenobarbital prescribed to prevent convulsions

• Dose levels need to be monitored carefully

• Concentrations high enough to control seizures without drowsiness

• Abuse potential high for short-acting barbiturates

• Positively reinforcing (rat studies, comparable to cocaine)

• Abuse peaked in the 1950s and 1960s, overshadowed by other illicit substances

• Less widely available as prescription drugs

• Stricter controls for obtaining excessive doses at pharmacies

• Doctors reluctant to prescribe due to suicide risk

Benzodiazepines

• Anti-anxiety medication

• Selective effect on anxiety

– Tranquilizing effects, rather than sedative effects

• Useful for anxiety and stress-related problems, but not the miracle drug promoted to be in the 1960’s/1970’s

• Long-acting – Valium (diazepam)

• Intermediate acting – Ativan (lorazepam), Restoril (temazepam)

• Short-acting – Xanax (alprazolam)

• Absorbed slowly into the bloodstream

– Relaxant effects develop more gradually than barbiturates à last longer

• Higher level of safety

– Respiratory centers in the brain not affected

– Rare to die of respiratory failure from overdose (intentional or accidental)

• As long as other depressant drugs not taken concurrently

• Dangers – long-acting benzodiazepines, rate of elimination slow, buildup after several doses (mostly in the elderly population)

– Drug-induced dementia in the elderly

– Long-acting benzodiazepine no longer recommended for this age group, switch to shorter-acting for anxiolytic effect

• Tolerance effects or acquired dependence

• Tolerance to the sedative effects (when taken for insomnia) – more required to induce sleep in later administrations

• Symptoms of benzodiazepine dependence:

– For slow-acting benzodiazepines withdrawal effect appear later (3-6 days)

– Anxiety level higher (worse than the original level; rebound anxiety)

– Insomnia, restlessness, agitation

– Less severe than barbiturate withdrawal

– Occur only after long-term use

– Subside in 1 – 4 weeks

In the Brain

• Heightens the effect of neurotransmitter GABA (gamma-aminobutyric acid)

• GABA exerts an inhibitory effect on the nervous system

• Greater inhibition with benzodiazepines, ↓activity level of neurons involved

• Cross-tolerance!

• Does not present the same potential for abuse as cocaine, alcohol or barbiturates

1. Weak reinforcers of behavior

– Rat studies, self-administration far less than for barbiturates

2. Slow onset of effect – no sudden “rush” felt in other drugs of abuse: cocaine, heroin, amphetamines

• Multiple substance abuse/polydrug abuse

– Alcoholics – to relax, avoid the smell of alcohol in breath

– Heroin abusers – augment euphoria, reduce anxiety when opiate levels fall

– Cocaine abusers – soften the crashing feeling when drugs wear off

• Misuse > abuse

– Most frequently prescribed and for excessive dosages

• Safe for short-term use, adverse side effects in long-term use

Rohypnol (flunitrazepam) – previously colorless, odorless, tasteless. Now turns blue when dissolved in clear liquid

Psychiatric drugs

• Used to treat mental illnesses (psychotropic)

• Schizophrenia (antipsychotic drugs) and mood disorders (depression, mania) (anti-depressants, mood stabilizers)

• Schizophrenia

– “Split-mind” – split off or broken off from a sense of reality.

– NOT multiple personality disorder (dissociative disorder)

– Delusions, leading to feelings of persecution, paranoia, auditory hallucinations – “voices”

– Not all suffer delusions, hallucinations – dulled emotions, catatonia (odd rigid, prolonged body posture)

– Early treatment: barbiturates, prefrontal lobotomies

Antipsychotic drugs

• Therapeutic medications:

• First-generation antipsychotics

– Chlorpromazine (Thorazine), haloperidol (Haldol)

– Effective in reducing symptoms for many patients

– But also carry the potential for development of severe movement-related motor problems (Parkinson’s-like symptoms)

• Second generation

– Clozapine (Clozaril), risperidone (Risperdal)

– Treat a wider spectrum of symptoms, without movement difficulties

– Clozaril – Agranulocytosis, lethal blood disease, need to monitor blood

– Risperdal – hyperglycemia, not recommended to treat psychotic symptoms related to dementia in the elderly – heart problems and respiratory infections

• Third generation

– Apiprazadole (Abilify)

– Effective treatment without risks from previous generation antispsychotics

– Modulate dopamine activity, stabilizes dopamine receptors

Antidepressants

• Major depression (severe, debilitating depression) most common form of mood disorder

• Emotional state far beyond ordinary feelings of sadness, grief or remorse

• Many depressed individuals turn to alcohol for relief

– Depressant action on the nervous system makes the condition worse

– Alcohol dependence and alcoholism

• Risk of suicide, major concern

– Attempts increased during the upswing of mood after a deep period of depression

– When depression is most intense, little energy to engage in suicidal feelings or thoughts

• First-generation antidepressants

– MAO (monoamine oxidase) inhibitors

• First group developed to treat depression

• Needed to be on a restricted diet to avoid serious adverse side effects (MAO breaks down tyramine)

– Tricyclic antidepressants

• Do not require dietary restrictions

• Effects on the cardiovascular system (elevated heart rate) make them undesirable for certain patients (cardiovascular disease)

• Second generation

– Fluoxetine (Prozac)

– Slow reuptake of serotonin

– Selective serotonin reuptake inhibitors (SSRIs)

• Third generation

– Slow reuptake of serotonin and norepinephrine in the brain

Mood Stabilizers

• Mania – as disruptive as depression

– Sleeplessness, impulsiveness, irritability, feelings of grandeur

• Bipolar disorder (manic-depression)

– Extreme mood swings back and forth between depression and mania

• Treatment

– Lithium carbonate or valproate (Depakote)

– Antipsychotic medications risperidone (Risperdal)

– Combination of antipsychotic and antidepressant medications (Symbyax)